Search results for "HLA-A1 Antigen"

showing 4 items of 4 documents

Complete congenital heart block in autoimmune hepatitis (SLA-positive).

1994

Complete congenital heart block is a serious complication of neonatal lupus erythematosus which most often occurs in children of mothers suffering from connective tissue disease. We report the occurrence of complete congenital heart block associated with autoimmune hepatitis (SLA-positive). A 32-year-old woman was treated for more than 10 years for autoimmune hepatitis (SLA-/ANA-positive) and remained in clinical remission under immunosuppressive therapy. She showed an MHC-haplotype typical for autoimmune hepatitis (A1, B8, DR3). After a normal first pregnancy, an emergency caesarean section was performed in the 32nd week of her second pregnancy because of fetal bradycardia. The child died …

AdultPediatricsmedicine.medical_specialtyHeart diseaseHeart blockAutoimmune hepatitisAutoantigensAutoimmune DiseasesHLA-B8 AntigenHepatitisHLA-DR3 AntigenRNA Small CytoplasmicmedicineHumansNeonatal lupus erythematosusHLA-A1 AntigenAutoimmune diseaseHepatitisPregnancyHepatologybusiness.industrymedicine.diseaseConnective tissue diseaseHeart BlockHaplotypesRibonucleoproteinsImmunologyChronic DiseaseFemalebusinessJournal of hepatology
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A neoepitope generated by an FLT3 internal tandem duplication (FLT3-ITD) is recognized by leukemia-reactive autologous CD8+ T cells.

2006

Abstract The FLT3 receptor tyrosine kinase is expressed in more than 90% of acute myelogeneous leukemias (AMLs), up to 30% of which carry an internal tandem duplication (ITD) within the FLT3 gene. Although varying duplication sites exist, most FLT3-ITDs affect a single protein domain. We analyzed the FLT3-ITD of an AML patient for encoding HLA class I–restricted immunogenic peptides. One of the tested peptides (YVDFREYEYY) induced in vitro autologous T-cell responses restricted by HLA-A*0101 that were also detectable ex vivo. These peptide-reactive T cells recognized targets transfected with the patient's FLT3-ITD, but not wild-type FLT3, and recognized the patient's AML cells. Our results …

FLT3 Internal Tandem DuplicationMyeloidmedicine.medical_treatmentImmunologyAntigen presentationMolecular Sequence DataHuman leukocyte antigenBiologyCD8-Positive T-LymphocytesIn Vitro TechniquesTransfectionBiochemistryCell LineEpitopesfluids and secretionshemic and lymphatic diseasesCell Line TumorGene DuplicationGene duplicationmedicineCytotoxic T cellHumansAmino Acid SequenceRNA MessengerHLA-A1 AntigenAntigen PresentationHLA-A Antigenshemic and immune systemsCell BiologyHematologyImmunotherapymedicine.diseaseMolecular biologyLeukemiaLeukemia Myeloid Acutemedicine.anatomical_structurefms-Like Tyrosine Kinase 3embryonic structurespsychological phenomena and processesBlood
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Identification of T cell epitopes by the use of rapidly generated mRNA fragments.

2004

Abstract Although the number of defined T cell epitopes of clinically relevant antigens is constantly increasing, there is still an enormous need to identify further peptides, processed from new antigens or presented by rare HLA molecules, respectively. Here we introduce a novel two-step approach for the rapid identification of T cell epitopes. It was established in the CMV infection model. From the peripheral blood of healthy donors sharing HLA-A1 according to HLA serotyping we isolated CD8 + T lymphocytes and generated dendritic cells (DCs). DCs were electroporated with CMV pp65 mRNA and tested for recognition by autologous CD8 + T lymphocytes in IFN-γ ELISPOT assays. In all 10 CMV-seropo…

T cellImmunologyEpitopes T-LymphocyteStreptamerHuman leukocyte antigenBiologyCD8-Positive T-LymphocytesEpitopeViral Matrix ProteinsAntigenmedicineImmunology and AllergyCytotoxic T cellHumansRNA MessengerHLA-A1 AntigenAntigen PresentationELISPOTDendritic CellsPhosphoproteinsVirologyMolecular biologymedicine.anatomical_structurePeptidesCD8Epitope MappingJournal of immunological methods
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HIV-1 Adaptation to Antigen Processing Results in Population-Level Immune Evasion and Affects Subtype Diversification

2014

Summary The recent HIV-1 vaccine failures highlight the need to better understand virus-host interactions. One key question is why CD8+ T cell responses to two HIV-Gag regions are uniquely associated with delayed disease progression only in patients expressing a few rare HLA class I variants when these regions encode epitopes presented by ∼30 more common HLA variants. By combining epitope processing and computational analyses of the two HIV subtypes responsible for ∼60% of worldwide infections, we identified a hitherto unrecognized adaptation to the antigen-processing machinery through substitutions at subtype-specific motifs. Multiple HLA variants presenting epitopes situated next to a giv…

T cellT-LymphocytesPopulationMolecular Sequence DataPopulationHIV InfectionsHuman leukocyte antigenBiologyGeneral Biochemistry Genetics and Molecular BiologyEpitopeArticleAfrica Southern03 medical and health sciencesEpitopesImmune systemGene FrequencymedicineHumansAmino Acid Sequenceeducationlcsh:QH301-705.5HLA-A1 Antigen030304 developmental biologyImmune EvasionGenetics0303 health scienceseducation.field_of_studyAntigen processingImmunogenicity030302 biochemistry & molecular biologyAdaptation Physiological3. Good healthEuropemedicine.anatomical_structurelcsh:Biology (General)HIV-1CD8Cell Reports
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